Thyroid follicular cell

Target gland or tissue · NIS TPO Thyroglobulin

TSH drives iodide trapping by the sodium-iodide symporter, then thyroid peroxidase oxidises and organifies iodide onto thyroglobulin tyrosines and couples them; colloid is endocytosed and hydrolysed to release T4 and T3.

Traced from the start

  1. Thyrotrophin-releasing hormone (paraventricular nucleus)
  2. Thyrotroph — thyroid-stimulating hormone
  3. Thyroid follicular cell

Detail

Output
About 100 microgram of T4 but only 5-10 microgram of T3 a day — the gland is a T4 factory and most T3 is made peripherally
Autoregulation
A large iodide load transiently blocks organification (Wolff-Chaikoff); in a nodular gland the same load can precipitate thyrotoxicosis (Jod-Basedow)
Drugs
Carbimazole and propylthiouracil block thyroid peroxidase; PTU also blocks peripheral T4 to T3 conversion, so it is preferred in thyroid storm and the first trimester
Uptake pathway
Radioiodine and pertechnetate enter through the same symporter, which is what an uptake scan measures

When it goes wrong

TSH-receptor stimulating antibody (Graves disease)

Thyrotoxicosis with diffuse goitre and suppressed TSH; the receptor is driven regardless of feedback, and the antibody crosses the placenta to cause neonatal thyrotoxicosis

Amiodarone or iodinated contrast

Type 1 thyrotoxicosis from excess substrate in a nodular gland, or type 2 destructive thyroiditis, which is treated with glucocorticoids rather than thionamides

Practise this structure

4 questions in the bank tagged Thyroid.