Explaining patient-controlled analgesia before a laparotomy - Hospital ward

CurtinYear 510 min + 2 min readingSurgery & perioperativeStandardPre-internship
  • Medication counselling

Ready

2 min reading, 10 min station, warning at 2 min

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Station - Instructions for candidate

You are an intern on the surgical ward of a metropolitan public hospital.

Thanh Le, a 61-year-old long-haul truck driver, is on the surgical ward the evening before a laparotomy booked for tomorrow morning for a bowel obstruction that has not settled. The nursing staff have mentioned a patient-controlled analgesia (PCA) pump with morphine for after the operation, and the patient has asked for it to be explained tonight.

Baseline observations (18:00)Comfortable at rest; afebrile. HR 78 regular, BP 126/74 mmHg, RR 14 breaths/min, SpO2 98% on room air, temperature 36.8 C. Pain 2/10 at rest and 5/10 on coughing.
Nursing handoverNil by mouth from midnight; consent signed; anti-embolic stockings applied; admission bloods sent and a group and save held. The PCA pump is prescribed for after surgery and will be set up in recovery.

Your tasks are to:

  1. Explain to the patient how the patient-controlled analgesia pump works, how to use it and the safety limits built into it.
  2. Counsel the patient on the common and serious side effects and on what to report to the nursing staff immediately.
  3. Explore and respond to the patient's concerns about dependence, and answer the question about whether someone else can press the button.
  4. Discuss what happens next: how the pump will be reviewed, how pain relief will continue afterwards, and when driving and work can be reconsidered.
  • You do NOT need to examine the patient.
  • You do not need to take a full history.
  • You do NOT need to prescribe, document or summarise to the examiner.
  • The observation chart and admission bloods are in the notes; the examiner will provide them if you ask.

This is a 10-minute station, with 2 minutes of reading time.

Generated by AI for practice; check doses and pathways against eTG / AMH before relying on them.