Not sleeping, talking fast and spending - Emergency Department

CurtinYear 515 min + 2 min readingMental healthChallengingPre-internship
  • Focused history
  • Dx + differentials
  • Initial Mx
  • Mental state examination

Ready

2 min reading, 15 min station, warning at 2 min

Focus the timer and press Space to start or pause.

Station - Instructions for candidate

You are an intern working in the Emergency Department of a metropolitan hospital in Perth. It is 9 am.

Jess Whitford, 34, has been brought in by her partner, Tom, who is worried that she has barely slept for over a week. Jess says there is nothing wrong with her. Tom is in the waiting room. She has agreed to 'a quick chat'.

Triage note34 F, brought by partner. Not sleeping for 10 days. Restless, over-familiar, no aggression. Says she is fine and wants to leave. Category 3.
ObservationsHR 104 /min, BP 134/84 mmHg, T 37.0 degrees C, RR 18 /min, SpO2 99% on room air, bedside glucose 5.6 mmol/L
GP summary (from the shared health record)Depressive episode 2023, sertraline 6 months, recovered. Sertraline 50 mg restarted 6 weeks ago for low mood; increased to 100 mg 3 weeks ago. No other medicines. No allergies.

Your tasks are to:

  1. Take a focused psychiatric history from Jess, including her medicines, substance use and a screen for medical causes.
  2. Present the mental state findings you have observed during the interview, and your assessment of risk, to the examiner.
  3. State your most likely diagnosis and the differential diagnoses to the examiner.
  4. Outline your initial management plan to the examiner, including how you would respond to Jess wanting to leave.
  • You do NOT need to perform a physical examination, and you do not need to summarise the history to the examiner - only the mental state findings and risk.
  • Collateral history from Tom is available from the examiner on request.
  • State drug, dose and route where relevant.

This is a 15-minute station, with 2 minutes of reading time.

Generated by AI for practice; check doses and pathways against eTG / AMH before relying on them.